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antibodies against ed1  (Bio-Rad)


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    Bio-Rad antibodies against ed1
    Antibodies Against Ed1, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 96/100, based on 3028 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/antibody+against+ed1/Mouse+anti+Rat+CD68/pmc11656461-102-13-19
    Average 96 stars, based on 3028 article reviews
    antibodies against ed1 - by Bioz Stars, 2026-09
    96/100 stars

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    Related Articles

    Incubation:

    Article Title: Human Umbilical Cord Mesenchymal Stem Cells Promote Anti-Inflammation and Angiogenesis by Targeting Macrophages in a Rat Uterine Scar Model.
    Article Snippet: HOXA10 Homeobox A10 LIF Leukemia inhibitory factor Arg1 Arginase 1 TNF-α Tumor necrosis factor alpha PBS Phosphate buffer CS Cesarean scar CSD Cesarean scar defect IUA Intrauterine adhesion PMA Phorbol 12-myristate 13-acetate LPS Lipopolysaccharide IFN-γ Interferon gamma IL4 Interleukin 4 IL13 Interleukin 13 HE Hematoxylin–eosin IHC Immunohistochemical Abbreviations MSCs Mesenchymal stem cells hUC-MSCs Human umbilical cord mesenchymal stem cells iNOS Inducible nitric oxide synthase IL10 Interleukin 10 VEGF Vascular endothelial growth factor

    Marker:

    Article Title: Alpha8beta1 integrin is upregulated in myofibroblasts of fibrotic and scarring myocardium.
    Article Snippet: Integrins mediate cell attachment to the extracellular matrix (ECM) regulating migration, proliferation, and differentiation.. We previously reported the presence of a8b1 integrin on cultured cardiac fibroblasts.. Extending this information, we localized a8b1 integrin in normal rat myocardial tissue, and investigated its expression pattern in rats chronically infused with angiotensin II (Ang II, 500 ng/kg/min), a well-recognized profibrotic factor. a8b1-integrin expression was analyzed by binding assay, western blotting, and immunohistochemistry.



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    Infiltration of macrophages in the brainstem after blast exposure. ED1 + cells (arrow heads) were observed exclusively in the meninges but not parenchyma of sham rats (A & E), whereas a significant number of ED1 + cells emerged in the parenchyma at 14 (B & F) and 29 days (C & G) after blast. By day 56, the number of parenchyma ED1 + cells was markedly reduced. Double-immunostaining revealed that some ED1 + macrophages are closely associated with CD31 + endothelial cells (arrows). Scale bars: 500 µm in A–D, 100 µm in E–H, 50 µm in I–L.

    Journal: Neural Regeneration Research

    Article Title: Traumatic brain injury induced by exposure to blast overpressure via ear canal

    doi: 10.4103/1673-5374.314311

    Figure Lengend Snippet: Infiltration of macrophages in the brainstem after blast exposure. ED1 + cells (arrow heads) were observed exclusively in the meninges but not parenchyma of sham rats (A & E), whereas a significant number of ED1 + cells emerged in the parenchyma at 14 (B & F) and 29 days (C & G) after blast. By day 56, the number of parenchyma ED1 + cells was markedly reduced. Double-immunostaining revealed that some ED1 + macrophages are closely associated with CD31 + endothelial cells (arrows). Scale bars: 500 µm in A–D, 100 µm in E–H, 50 µm in I–L.

    Article Snippet: To determine if macrophages extravasated into the brain, double-immunofluorescence were performed using antibodies against macrophage marker ED1 (Cat#MAB1435, 1:400; EMD Millipore) and endothelial cell marker CD31 (Cat# PA5-16301, 1:200; Thermo Fisher Scientific, Waltham, MA, USA).

    Techniques: Double Immunostaining